___

• 1]. Snesky TE, Liu DT. Endometriosis: associations with menorrhagia, infertility, and oral contraceptives. Int J Gynaecol Obstet 1980; 17: 573–76.
• [2]. Houston DE. Evidence for the risk of pelvic endometriosis by age, race, and socioeconomic status. Epidemiol Rev 1984; 6: 167–91.
• [3]. Cramer DW. Epidemiology of endometriosis in adolescents. In: Wilson EA, ed. Endometriosis. New York: Alan Liss, 1987: 5–8.
• [4] Linda C. Giudice, Lee C. Kao. Endometriosis. Lancet 2004; 364: 1789-1799.
• [5] Gruenwald, P. 1942. Origin of endometriosis fromthe mesenchyme of the coelomic walls. Am. J. Obstet. Gynecol. 44: 474.
• [6]. Gardner,G.H. et al. 1953. Endometriosis. Obstet.Gynecol. 1: 637.
• [7]. Schrodt, G.R. et al. 1980. Endometriosis of the male urinary system: a case report. J. Urol. 124: 723.
• [8]. Oliker A.J., Harris A.E.. 1971. Endometriosis of the bladder in a male patient. J. Urol. 106: 859.
• [9]. Clark A.H. 1948. Endometriosis in a young girl. JAMA 136: 690.
• [10]. Schifrin B.S. et al. 1973. Teenage endometriosis. Am. J. Obstet. Gynecol. 116: 980.
• [11]. Hobbs J.E., Bortnick A.R.. 1940. Endometriosis of the lung: an experimental and clinical study. Am. J. Obstet. Gynecol. 40: 843.
• [12]. Cassina P.C. et al. 1997. Catamenial hemoptysis: diagnosis with MRI. Chest 111: 1450.
• [13]. Van Schil P.E. et al. 1996. Catamenial pneumothorax caused by thoracic endometriosis. Ann. Thorac. Surg. 62: 586.
• [14]. Olive D.L., Henderson D.Y.. 1987. Endometriosis and mullerian anomalies. Obstet. Gynecol. 69: 415.
• [15]. R. Gazvani, A. Templeton. New considerations for the pathogenesis of endometriosis. International Journal of Gynecology & Obstetrics 2002; 76: 117-126.
• [16]. D. Vinatier, G. Orazi, M. Cosson, P. Dufour. Theories of endometriosis. Europena Journal of Obstetrics & Gynecology and Reproductive Biology 2001; 96: 21-34.
• [17]. Burney RO, Giudice LC. Pathogenesis and pathophysiology of endometriosis. Fertil Steril. 2012 Sep;98(3):511-9
• [18]. Levander G, Normann P. The pathogenesis of endometriosis; an experimental study. Acta Obstet Gynecol Scand 1955;34:366–98.
• [19]. Merrill JA. Endometrial induction of endometriosis across Millipore filters. Am J Obstet Gynecol 1966;94:780–90.
• [20]. Matsuura K, Ohtake H, Katabuchi H, Okamura H. Coelomic metaplasia theory of endometriosis: evidence from in vivo studies and an in vitro experimental model. Gynecol Obstet Invest. 1999;47 Suppl 1:18-20; discussion 20-2
• [21]. Russell W. Aberrant portions of the mullerian duct found in an ovary. Ovarian cysts of mullerian origin. Bull Johns Hopkins Hosp 1899;10:8.
• [22]. Missmer SA, Hankinson SE, Spiegelman D, Barbieri RL, Michels KB, Hunter DJ. In utero exposures and the incidence of endometriosis. Fertil Steril 2004;82:1501–8.
• [23]. Leibovic DI, Mueller MD, Taylor RN. Immunobiology of endometriosis. Fertil Steril 2001; 75: 1–10.
• [24].Koninckx PR, Kennedy SH, Barlow DH. Endometriotic disease: the role of the peritoneal fluid. Hum Reprod Update 1998; 4: 741–51.
• [25]. Sharpe-Timms KL, Piva M, Ricke EA, Surewicz K, Zhang YL, Zimmer RL. Endometriosis synthesizes and secretes a haptoglobin-like protein. Biol Reprod 1998; 58: 988 94.
• [26]. Sharpe-Timms KL, Zimmer RL, Ricke EA, Piva M, Horowitz GM. Endometriotic haptoglobin binds to peritoneal macrophages and alters their function in women with endometriosis. Fertil
• Steril 2002; 78: 810–19. [27]. Piva M, Horowitz GM, Sharpe-Timms KL. Interleukin-6 differentially stimulates haptoglobin production by peritoneal and endometriotic cells in vitro: a model for endometrium peritoneum interaction in endometriosis. J Clin Endocrinol Metab 2001; 86: 2553–61.
• [28]. Oosterlynck DJ, Cornillie FJ, Waer M, Vandeputte M, Koninckx PR. Women with endometriosis show a defect in natural killer activity resulting in a decreased cytotoxicity to autologous endometrium. Fertil Steril 1991; 56: 45–51.
• [29]. Chegini N. Peritoneal molecular environment, adhesion formation, and clinical implication. Front Biosci 2002; 1: e91–115.
• [30]. Gazvani R, Templeton A. Peritoneal environment, cytokines and angiogenesis in the pathophysiology of endometriosis. Reproduction 2002; 123: 217–26.
• [31]. Bedaiwy MA, Falcone T. Peritoneal fluid environment in endometriosis: clinicopathological implications. Minerva Ginecol 2004; 55: 333–45.
• [32]. Zondervan KT, Cardon LR, Kennedy S. What makes a good case control study? Design issues for complex traits such as endometriosis. Hum Reprod 2002; 17: 1415–23.
• [33]. Nothnick WB. Treating endometriosis as an autoimmune disease. Fertil Steril 2001; 76: 223–40
• [34]. Blumenthal RD, Samoszuk M, Taylor AP, Brown G, Alisauskas R, Goldenberg DM. Degranulating eosinophils in human endometriosis. Am J Pathol 2000; 156: 1581–88.
• [35]. Hornung D, Dohrn K, Sotlar K, et al. Localization in tissues and secretion of eotaxin by cells from normal endometrium and endometriosis. J Clin Endocrinol Metab 2000; 85: 2604–08.
• [36]. Sinaii N, Cleary SD, Ballweg ML, Nieman LK, Stratton P. High rates of autoimmune and endocrine disorders, fibromyalgia, chronic fatigue syndrome, and atopic diseases among women with endometriosis: a survey analysis. Hum Reprod 2002; 17: 2715–24.
• [37].Halban J. Metastatic hysteroadenosis. Wien klin Wochenschr 1924;37:1205–6.
• [38].Sampson JA. Metastatic or embolic endometriosis, due to the menstrual dissemination of endometrial tissue into the venous circulation. Am J Pathol 1927;3:93–110.
• [39]. Sasson IE, Taylor HS. Stem cells and the pathogenesis of endometriosis. Ann N Y Acad Sci. 2008 Apr;1127:106-15. doi: 10.1196/annals.1434.014. Review.
• [40]. Reid G.D. et al. 2003. Hepatic endometriosis: a case report and review of the literature. Authorst. N. Z. J. Obstet.Gynaecol. 43: 87–89.
• [41]. Simpson JL, Elias S, Malinak LR, Buttram VC Jr. Heritable aspects of endometriosis, I: genetic studies. Am J Obstet Gynecol 1980; 137: 327–31.
• [42]. Kennedy S, Mardon H, Barlow D. Familial endometriosis. J Assist Reprod Genet 1995; 12: 32–34.
• [43]. Moen MH. Endometriosis in monozygotic twins. Acta Obstet Gynecol Scand 1994; 73: 59–62.
• [44]. Hadfield RM, Mardon HJ, Barlow DH, Kennedy SH. Endometriosis in monozygotic twins. Fertil Steril 1997; 68: 941–42.
• [45]. Treloar SA, O’Connor DT, O’Connnor VM, Martin NG. Genetic influences on endometriosis in an Australian twin sample. Fertil Steril 1997; 71: 701–10.
• [46]. dos Reis RM et al (1999) Familial risk among patients with endometriosis. J Assist Reprod Genet 16(9):500–503
• [47]. Lamb KT (1986) National data registry for endometriosis. Wis Med J 85(8):27–28
• [48]. Kennedy S, Bennett S, Weeks DE. Affected sib-pair analysis in endometriosis. Human Reprod Update 2001; 7: 411–18.
• [49]. Kennedy SH. Genetics of endometriosis: a review of the positional cloning approaches. Sem Reprod Med 2003; 21: 111–18.
• [50]. Bischoff FZ, Simpson JL. Heritability and molecular genetic studies in endometriosis. Hum Reprod Update 2000; 6: 37–44.
• [51]. Bischoff FZ, Marquez-Do Dom Dang D, Carson SA, Buster JE, Simpson JL. NAT2 and GST M1 DNA polymorphisms: increased GSTM1 (active) genotype in endometriosis. Fertil Steril 2002; 77 (suppl 1): S17.
• [52]. Nakago S, Hadfield RM, Zondervan KT, et al. Association between endometriosis and N acetyl transferase polymorphisms in a UK population. Mol Hum Reprod 2001; 7: 1079–82.
• [53]. Baxter SW, Thomas EJ, Campbell IG. GSTM1 null polymorphism and susceptibility to endometriosis and ovarian cancer. Carcinogenesis 2001; 22: 63–5.
• [54]. Zondervan KT, Cardon LR, Kennedy S. What makes a good case control study? Design issues for complex traits such as endometriosis. Hum Reprod 2002; 17: 1415–23.
• [55].E. Attar E, Tokunaga H, Imir G, Yilmaz MB, Redwine D, Putman M, Gurates B, Attar R, Yaegashi N, Hales DB, Bulun SE. Prostaglandin E2 via steroidogenic factor-1 coordinately regulates transcription of steroidogenic genes necessary for estrogen synthesis in endometriosis. J Clin Endocrinol Metab. 2009 Feb;94(2):623-31.
• [56]. Georgiou I et al (1999) Association of estrogen receptor gene polymorphisms with endometriosis. Fertil Steril 72(1):164–166
• [57]. Hsieh YY et al (2001) Androgen receptor trinucleotide polymorphism in endometriosis. Fertil Steril 76(2):412–413
• [58]. Kitawaki J et al (2001) Oestrogen receptoralpha gene polymorphism is associated with endometriosis, adenomyosis and leiomyomata. Hum Reprod 16(1):51–55
• [59]. Chang CC et al (2002) The proline form of p53 codon 72 polymorphism is associated with endometriosis. Fertil Steril 77(1):43–45
• [60]. Kado N et al (2002) Association of the CYP17 gene and CYP19 gene polymorphisms with risk of endometriosis in Japanese women. Hum Reprod 17(4):897–902
• [61]. Wieser F et al (2003) Analysis of an interleukin-6 gene promoter polymorphism in women with endometriosis by pyrosequencing. J Soc Gynecol Investig 10(1):32–36
• [62]. Fujimoto J et al (1999) Expression of oestrogen receptor-alpha and -beta in ovarian endometriomata. Mol Hum Reprod 5(8):742–747
• [63]. Luisi S et al (2006) Estrogen receptor gene polymorphisms are associated with recurrence of endometriosis. Fertil Steril 85(3): 764–766
• [64]. Kim SH et al (2005) Estrogen receptor dinucleotide repeat polymorphism is associated with minimal or mild endometriosis. Fertil Steril 84(3):774–777
• [65]. Renner SP et al (2006) Evaluation of clinical parameters and estrogen receptor alpha gene polymorphisms for patients with endometriosis. Reproduction 131(1):153–161
• [66]. Wu CH et al (2012) Polymorphisms of dioxin receptor complex components and detoxification-related genes jointly confer susceptibility to advanced-stage endometriosis in the Taiwanese Han population. Am J Reprod Immunol 67(2):160–168
• [67]. Attar R, Agachan B, Kucukhuseyin O, Toptas B, Attar E, Isbir T. Association of interleukin 1beta gene (+3953) polymorphism and severity of endometriosis in Turkish women. Mol Biol Rep. 2010 Jan;37(1):369-74
• [68]. Kajihara H et al (2011) New insights into the pathophysiology of endometriosis: from chronic inflammation to danger signal. Gynecol Endocrinol 27(2):73–79
• [69]. Latha M et al (2011) Molecular pathogenesis of endometriosis; Toll-like receptor-4 A896G (D299G) polymorphism: a novel explanation. Genet Test Mol Biomark 15(3):181–184
• [70]. Han YJ et al (2009) Haplotype analysis of the matrix metalloproteinase- 9 gene associated with advanced-stage endometriosis. Fertil Steril 91(6):2324–2330
• [71]. Borghese B et al (2008) Genetic polymorphisms of matrix metalloproteinase 12 and 13 genes are implicated in endometriosis progression. Hum Reprod 23(5):1207–1213
• [72]. Cramer DW et al (1996) Endometriosis associated with the N314D mutation of galactose-1- phosphate uridyl transferase (GALT). Mol Hum Reprod 2(3):149–152
• [73]. Baranova H et al (1999) Possible involvement of arylamine N-acetyltransferase 2, glutathione S-transferases M1 and T1 genes in the development of endometriosis. Mol Hum Reprod 5(7):636–641
• [74]. Munksgaard PS, Blaakaer J (2012) The association between endometriosis and ovarian cancer: a review of histological, genetic and molecular alterations. Gynecol Oncol 124(1):164 169
• [75]. Martini M et al (2002) Possible involvement of hMLH1, p16(INK4a) and PTEN in the malignant transformation of endometriosis. Int J Cancer 102(4):398–406
• [76]. Kobayashi H et al (2009) Molecular pathogenesis of endometriosis- associated clear cell carcinoma of the ovary (review). Oncol Rep 22(2):233–240
• [77]. Fanton JW, Golden JG. Radiation-induced endometriosis in Maccaca mulatta. Radiat Res 1991; 126: 141–46.
• [78]. Rier SE, Martin DC, Bowman RE, et al. Endometriosis in rhesus monkeys (Maccaca mulatta) following chronic exposure to 2,3,7,8 tetrachlorodibenzo-p-dioxin. Fundam Appl Toxicol 1993; 21: 431–41.
• [79]. Koninckx PR, Braet P, Kennedy SH, et al. Dioxin pollution and endometriosis in Belgium. Hum Reprod 1994; 91001–02.
• [80]. Pauwels A, Schepens PJ, D’Hooghe T, Delbeke L, Dhont M, Brouwer A, Weyler J. The risks of endometriosis and exposure to dioxins and polychlorinated biphenyls: a case-controlled study of infertile women. Hum Reprod 2001; 16: 2050–55.
• [81]. Eskenazi B, Mocarelli P, Warner M, et al. Serum dioxin concentrations and endometriosis: a cohort study in Seveso, Italy. Environ Health Perspect 2002; 110: 629–34.
• [82]. Myers JP, Guillette LJ Jr, Palanza P, Parmigiani S, Swan SH, von Saal FS. The emerging science of endocrine disruption. Science and Culture Series. International Seminar on Nuclear War and Planetary Emergencies. 28th Session, 2003, Erice, Italy.
• [83]. Stokstad E. Pollution gets personal. Science 2004; 304: 1892–94.
• [84]. Sampson JA, Peritoneal endometriosis due to the menstrual dissemination of endometrial tissue into the peritoneal cavity, Am J Obstet Gynecol 14:422, 1927.
• [85]. Halme J, Hammond MG, Hulka JF, Raj SG, Talbert LM, Retrograt menstruation in healthy women and in patients with endo-metriosis, Obstet Gynecol 64:151, 1984.
• [86]. Liu DTY, Hitchchock A, Endometriosis: its association with retrograt menstruation, dysmenorrhea, and tubal pathology, Br J Obstet Gynaecol 93:859, 1986.
• [87]. Blumenkrantz MJ, Gallagher N, Bashore RA, Tenckhoff H, Retrograt menstruation in women undergoing chronic peritoneal dialysis, Obstet Gynecol 57:667, 1981.
• [88]. Kruitwagen RFPM, Poels LG, Willemsen WNP, Jap PHK, Thomas CMG, Rolland R, Endometrial epithelial cells in perito neal fuid during the early follicular phase, Fertil Steril 55:297, 1991.
• [89]. Witz CA, Monotoya-Rodriguez IA, Schenken RS, Whole explants of peritoneum and endometrium: a novel model of the early endo-metriosis lesion, Fertil Steril 71:56, 1999.
• [90]. Maas JW, Groothuis PG, Dunselman GA, de Goeij AF, Struijker-Boudier HA, Evers JL, Development of endometriosis-like lesions after transplantation of human endometrial fragments onto the chick embryo chorioallantoic membrane, Hum Reprod 16:627, 2001.
• [91]. TeLinde R, Scott R, Experimental endomet riosis, Am J Obstet Gynecol 60:1147, 1950.
• [92]. Scott RB, TeLinde RW, Wharton Jr LR, Further studies on experimental endometriosis, Am J Obstet Gynecol 66:1082, 1953.
• [93]. D’Hooghe TM, Bambra CS, Raeymaekers BM, De Jonge I, Lauweryns JM, Koninckx PR, Intrapelvic injection of menstrual endometrium causes endometriosis in baboons (Papio cynocephalus and Papio anubis), Am J Obstet Gynecol 173:125, 1995
• [94]. Ridley JH & Edwards IK. Experimental endometriosis in the human. American Journal of
• Obstetrics and Gynecology 1958; 76: 783–790. [95]. D’Hooghe TM 1997. Clinical relevance of the baboon as a model for the study of endometriosis. Fertility Sterility 68: 625.
• [96]. Salamanca A, Beltran E. 1995. Subendometrial contractility in menstrual phase visualized by transvaginal sonography in patients with endometriosis. Fertility Sterility. 64: 195.
• [97]. Kruitwagen R F. Menstruation as a pelvic aggressor. Baillières Clin Obstet Gynaecol 1993; 7: 687–700.
• [98].Viganò P, Parazzini F, Somigliana E, Vercel lini P. Endometriosis: epidemiology and aetiological factors. Best Pract Res Clin Obstet Gynaecol. 2004 Apr;18(2):177-200. Review
• [99]. Garcia-Velasco J A, Arici A. Apoptosis and the pathogenesis of endometriosis. Semin Reprod Med 2004; 21: 165–78.
• [100]. Gebel HM, Braun DP, Tambur A et al. Spontaneous apoptosis of endometrial tissue is impaired in women with endometriosis. Fertility and Sterility 1998; 69: 1042–1047.
• [101]. Meresman GF, Vighi S, Buquet RA et al. Apoptosis and expression of Bcl-2 and Bax in eutopic endometrium from women with endometriosis. Fertility and Sterility 2000; 74: 760 766.
• [102]. Goumenou A, Panayiotides I, Matalliotakis I et al. Bcl-2 and Bax expression in human endometriotic and adenomyotic tissues. European Journal of Obstetrics, Gynecology and Reproductive Biology 2001; 99: 256–260.
• [103]. Watanabe H, Kanzaki H, Narakuwa S et al. Bcl-2 and Fas expressiopn in eutopic and ectopic human endometrium during the menstrual cycle in relation to endometrial cell apoptosis. American Journal of Obstetrics and Gynecology 1997; 176: 360–368.
• [104]. Jones RK, Searle RF & Bulmer JN. Apoptosis and bcl-2 expression in normal human endometrium, endometriosis and adenomyosis. Human Reproduction 1998; 13: 3496–3502.
• [105]. Bulen S E, Young S, Fang Z, Gurates B, Tamura M, Sebastian S. Estrogen production and metabolism in endometriosis. Ann NY Acad Sci 2002; 955: 75–85.
• [106]. Beliard A, Donnez J, Nisolle M & Foidart JM. Localization of laminin, fibronectin, E-cadherin, and integrins in endometrium and endometriosis. Fertility and Sterility 1997; 67: 266 272.
• [107]. Brooks PC, Sromblad S, Sanders LC et al. Localization of matrix metalloproteinase MMP-2 to the surface of invasive cells by interaction with integrin alphav beta3. Cell 1996; 85: 683–693.
• [108]. Collette T, Maheux R, Mailloux J et al. Increased expression of matrix metalloproteinase-9 in the eutopic endometrial tissue of women with endometriosis. Hum Reprod 2006; 21:3059–3067.
• [109]. Cox KE, Piva M, Sharpe-Timms KL. Differential regulation of matrix metalloproteinase-3 gene expression in endometriotic lesions compared with endometrium. Biol Reprod 2001; 65:1297–1303.
• [110]. Uzan C, Cortez A, Dufournet C et al. Eutopic endometrium and peritoneal, ovarian and bowel endometriotic tissues express a different profile of matrix metalloproteinases -2, -3 and -11, and of tissue inhibitor metalloproteinases-1 and -2. Virchows Arch 2004; 445:603–609.
• [111]. Salamonsen LA, Butt AR, Hammond FR et al. Production of endometrial matrix metalloproteinases, but not their tissue inhibitors, is modulated by progesterone withdrawal in an in vitro model for menstruation. Journal of Clinical Endocrinology and Metabolism 1997; 82: 1409–1415.
• [112]. Bruner KL, Eisenberg E, Gorstein F & Osteen KG. Progesterone and transforming growth factor-beta coordinately regulate suppression of endometrial matrix metalloproteinases in a model of experimental endometriosis. Steroids 1999; 64: 648–653.
• [113]. Bruner KL, Matrisian LM, Rodgers WH et al. Suppression of matrix metalloproteinases inhibits establishment of ectopic lesions by human endometrium in nude mice. Journal of Clinical Investigation 1997; 99: 2851–2857.
• [114]. Chung HW, Lee JY, Moon HS et al. Matrix metalloproteinase-2, membranous type 1 matrix metalloproteinase, and tissue inhibitor of metalloproteinase-2 expression in ectopic and eutopic endometrium. Fertility and Sterility 2002; 78: 787–795.
• [115]. Sharpe-Timms KL. Basic research in endometriosis. Obstetrics and Gynecology Clinics of North America 1997; 24: 269–290.
• [116]. Sharpe-Timms KL. Endometrial anomalies in women with endometriosis. Annales of New York Academy of Sciences 2001; 943: 131–147.
• [117]. Healy DL, Rogers PA, Hii L & Wingfield M. Angiogenesis: a new theory for endometriosis. Human Reproduction Update 1998; 4: 736–740.
• [118]. Donnez J, Smoes P, Gillerot S et al. Vascular endothelial growth factor (VEGF) in endometriosis. Human Reproduction 1998; 13: 1686– 1690.
• [119]. McLaren J, Prentice A, Charnock-Jones DS & Smith SK. Vascular endothelial growth factor (VEGF) concentrations are elevated in peritoneal fluid of women with endometriosis. Human Reproduction 1996; 11: 220–223.
• [120]. Tan XJ, Lang JH, Liu DY et al. Expression of vascular endothelial growth factor and thrombospondin-1 mRNA in patients with endometriosis. Fertility and Sterility 2002; 78: 148 153.
• [121]. Dechaud H, Craig A, Monotoya-Rodriguez IA et al. Mesothelial cell-associated hyaluronic acid promotes adhesion of endometrial cells to mesothelium. Fertility and Sterility 2001; 76: 1012–1018.
• [122]. Bulun, S.E., et al., Role of aromatase in endometrial disease. J Steroid Biochem Mol Biol., 2001. 79: p. 19-25.
• [123].Tsai, S.J., et al., Regulation of steroidogenic acute regulatory protein expression and progesterone production in endometriotic stromal cells. Journal of Clinical Endocrinology & Metabolism, 2001. 86: p. 5765-5773.
• [124]. Gurates, B., et al., WT1 and DAX-1 inhibit aromatase P450 expression in human endometrial and endometriotic stromal cells. J Clin Endocrinol Metab, 2002. 87: p. 4369-4377.
• [125]. Noble L.S., et al., Aromatase expression in endometriosis. Journal of Clinical Endocrinology and Metabolism, 1996. 81: p. 174-179.
• [126]. Noble L.S., et al., Prostaglandin E2 stimulates aromatase expression in endometriosisderived stromal cells. Journal of Clinical Endocrinology and Metabolism, 1997. 82: p. 600-606.
• [127]. Sun H.S., et al., Transactivation of steroidogenic acute regulatory protein in human endometriotic stromalcells is mediated by the prostaglandin EP2 receptor. Endocrinology, 2003. 144: p. 3934-3942.
• [128]. Wu, M., et al., Distinct Regulation of Cyclooxygenase-2 by Interleukin-1{beta} in Normal and Endometriotic Stromal Cells. J Clin Endocrinol Metab, 2004. 89.
• [129]. Ota, H., et al., Distribution of cyclooxygenase-2 in eutopic and ectopic endometrium in endometriosis and adenomyosis. Human Reproduction, 2001. 16: p. 561-566.
• [130]. Attia GR, Zeitoun K, Edwards D, Johns A, Carr BR, Bulun SE. Progesterone receptor isoform A but not B is expressed in endometriosis. J Clin Endocrinol Metab 2000; 85: 2897 902.
• [131]. Lessey BA, Metzger DA, Haney AF, McCarty KS Jr. Immunohistochemical analysis of estrogen and progesterone receptors in endometriosis: comparison with normal endometrium during the menstrual cycle and the effect of medical therapy. Fertil Steril 1989; 49: 229–35.
• [132]. Berqvist A, Ferno M. Oestrogen and progesterone receptors in endometriotic tissue and endometrium: comparison of different cycle phases and ages. Hum Reprod 1999; 8: 2211 17.
• [133]. Maruyama, T., H. Masuda, M. Ono, et al. 2010. Human uterine stem/progenitor cells: their possible role in uterine physiology and pathology. Reproduction 140: 11– 22.
• [134]. Patel, A.N., E. Park, M. Kuzman, et al. 2008. Multipotent menstrual blood stromal stem cells: isolation, characterization, and differentiation. Cell Transplant. 17: 303– 311.
• [135]. Gargett, C.E., Masuda H. 2010. Adult stem cells in the endometrium. Mol. Hum. Reprod. 16: 818–834.
• [136]. Chan,, R.W., Schwab K.E., Gargett C.E. 2004. Clonogenicity of human endometrial epithelial and stromal cells. Biol. Reprod. 70: 1738– 1750.
• [137].Gargett, C.E. 2004. Stem cells in gynaecology. Aust. N. Z. J. Obstet. Gynaecol. 44: 380–386.
• [138].16. Schwab K.E., Chan R.W., Gargett C.E.. 2005. Putative stem cell activity of human endometrial epithelial and stromal cells during the menstrual cycle. Fertil. Steril. 84(Suppl. 2): 1124–1130.
• [139]. Sasson I.E.,Taylor H.S.. 2008. Stemcells and the pathogenesis of endometriosis. Ann. N. Y. Acad. Sci. 1127: 106–115.
• [140]. Taylor, H.S. 2004. Endometrial cells derived from donor stem cells in bone marrow transplant recipients. JAMA 292: 81–85.
• [141]. Du, H, Taylor H.S.. 2007. Contribution of bone marrowderived stemcells to endometrium and endometriosis. Stem Cells 25: 2082–2086.
• [142]. Mints, M., M. Jansson, B. Sadeghi, et al. 2008. Endometrial endothelial cells are derived fromdonor stemcells in a bone marrow transplant recipient. Hum. Reprod. 23: 139–143.
• [143]. Ikoma,T., S.Kyo, Y.Maida, et al. 2009. Bone marrow-derived cells from male donors can compose endometrial glands in female transplant recipients. Am. J. Obstet. Gynecol. 201: e601–e608
• [144]. Masuda, H., T.Maruyama, E. Hiratsu, et al. 2007. Noninvasive and real-time assessment of reconstructed functional human endometrium in NOD/SCID/gamma c(null) immunodeficient mice. Proc. Natl. Acad. Sci. USA 104: 1925– 1930.