In this study, the kinetic, structural and functional changes of the ventriculus in dogs with myocardial infarction introduced experimentally were examined using M-Mode and two dimensional (2-D) echocardiography. 21 healthy, mixed-breed dogs of both genders (16 females and 5 males) and of various ages were used in this study. Different coronary arteries were ligated in each dog with silk sutures and the dogs were grouped accordingly: left distalis descending coronary artery (Group I, n=7), right distalis descending coronary artery (Group II, n=7) and both left and right distalis decending coronary artery (Group III, n=7). All the dogs were examined using echocardiography prior to the study. After the coronary arteries were ligated, this was repeated at 2, 4, 8 and 12-hour intervals for seven days. Wall motion abnormalites were observed in areas of infarction (left ventricle posterior wall = LVPW) in addition to irregularities in mitral valvule (MV) movements 2 hours after coronary ligation in Group I Increases were observed in the left ventricle (LV) end-systolic diameter (EDS) and end-diastolic diameter (EDD), circumference (C) and area (A); and also int he right ventricle (RV) EDS and RVEDD, RVC, RVA, RV systolic volumes (SV) and diastolic volumes (DV) in Group I, whereas the LV shortening fraction (SF), RVSF and ejection fraction (EF) (p

In this study, the kinetic, structural and functional changes of the ventriculus in dogs with myocardial infarction introduced experimentally were examined using M-Mode and two dimensional (2-D) echocardiography. 21 healthy, mixed-breed dogs of both genders (16 females and 5 males) and of various ages were used in this study. Different coronary arteries were ligated in each dog with silk sutures and the dogs were grouped accordingly: left distalis descending coronary artery (Group I, n=7), right distalis descending coronary artery (Group II, n=7) and both left and right distalis decending coronary artery (Group III, n=7). All the dogs were examined using echocardiography prior to the study. After the coronary arteries were ligated, this was repeated at 2, 4, 8 and 12-hour intervals for seven days. Wall motion abnormalites were observed in areas of infarction (left ventricle posterior wall = LVPW) in addition to irregularities in mitral valvule (MV) movements 2 hours after coronary ligation in Group I Increases were observed in the left ventricle (LV) end-systolic diameter (EDS) and end-diastolic diameter (EDD), circumference (C) and area (A); and also int he right ventricle (RV) EDS and RVEDD, RVC, RVA, RV systolic volumes (SV) and diastolic volumes (DV) in Group I, whereas the LV shortening fraction (SF), RVSF and ejection fraction (EF) (p