The correlation between the increase rate of serum creatininelevels and long-term adverse clinical outcomes in patientswith non st-segment elevation myocardial infarction

The correlation between the increase rate of serum creatininelevels and long-term adverse clinical outcomes in patientswith non st-segment elevation myocardial infarction

Aim: We purposed to evaluate the correlation between the rate of increase in SCrea levels and major adverse cardiac andcerebrovascular events(MACCE) in non ST-segment elevation myocardial infarction(NSTEMI) patients who was made coronaryangiography(CAG) in this study. According to studies on especially stable coronary artery disease (SCAD); contrast-induced acutekidney injury CI-AKI) is described as an rising in serum creatinine (SCrea) levels more than 0.5 mg / dl or more than 25% within 48-72hours after the contrast agent implementation. However, data on the increase rates of SCrea levels in patients with acute coronarysyndrome (ACS) are insufficient.. Materials and Methods: 884 NSTEMI patients were admitted to our study. We classified the patients into 3 groups according to theincrease rates in SCrea values; first group(∆SCrea

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  • 1. Mehran R, Aymong ED, Nikolsky E, et al. A simple risk score for prediction of contrast-induced nephropathy after percutaneous coronary intervention: development and initial validation. J Am Coll Cardiol 2004;44:1393-9.
  • 2. McCullough PA, Adam A, Becker CR, et al. CIN Consensus Working Panel. Epidemiology and prognostic implications of contrast-induced nephropathy. Am J Cardiol 200618;98:5-13.
  • 3. Kidney Disease: Improving Global Outcomes (KDIGO) AKI Work Group. KDIGO clinical practice guideline for acute kidney injury. Kidney Inter Suppl 2012;2:1–138.
  • 4. Morcos SK. Contrast medium-induced nephrotoxicity. In: Dawson P, Cosgrove DO, Grainger RG, editors. Textbook of Contrast Media. Oxford, England: Isis Medical Media Ltd 1999:135-48.
  • 5. Nikolsky E, Aymong ED, Dangas G, et al. Radio contrast nephropathy: identifying the high-risk patient and the implications of exacerbating renal function. Rev Cardiovasc Med 2003;4(suppl 1):7-14.
  • 6. Harjai KJ, Raizada A, Shenoy C, et al. A comparison of contemporary definitions of contrast nephropathy in patients undergoing percutaneous coronary intervention and a proposal for a novel nephropathy grading system. Am J Cardiol 2008;101:812-9.
  • 7. Leoncini M, Toso A, Maioli A, et al. Early high-dose rosuvastatin and cardioprotection in the protective effect of rosuvastatin and anti- platelet therapy on contrast-induced acute kidney injury and myocardial damage in patients with acute coronary syndrome (PRATO-ACS) study. Am Heart J 2014;168:792–7.
  • 8. Ruparelia N, Digby JE, Jefferson A, et al. Myocardial infarction causes inflammation and leukocyte recruitment at remote sites in the myocardium and in the renal glomerulus. Inflamm Res 2013;62:515–25.
  • 9. Narula A, Mehran R, Weisz G, et al. Contrast-induced acute kidney injury after primary percutaneous coronary intervention: results from the HORIZONSAMI substudy. Eur Heart J 2014;35:1533-40.
  • 10. Uzunhasan I, Yildiz A, Arslan S, et al. ContrastInduced Acute Kidney Injury Is Associated With LongTerm Adverse Events in Patients With Acute Coronary syndrome. Angiology 2017;68:621-6.
  • 11. Abaci O, Arat Ozkan A, Kocas C, et al. Impact of Rosuvastatin on contrast-induced acute kidney injury in patients at high risk for nephropathy undergoing elective angiography. Am J Cardiol 2015;115:867-71.
  • 12. Khwaja A. KDIGO clinical practice guidelines for acute kidney injury. Nephron Clin Pract 2012;120:c179-84.
  • 13. Mehta RL, Kellum JA, Shah SV, et al. Acute Kidney Injury Network. Acute Kidney Injury Network: report of an initiative to improve outcomes in acute kidney injury. Crit Care 2007;11:R31.
  • 14. Thygesen K, Alpert JS, Jaffe AS, et al. Joint ESC/ ACCF/AHA/WHF task force for third universal definition of myocardial infarction. J Am Coll Cardiol 2012;60:1581-98
  • 15. Levey AS, Coresh J, Greene T, et al. Chronic kidney disease epidemiology collaboration. expressing the modification of diet in renal disease study equation for estimating glomerular filtration rate with standardized serum creatinine values. Clin Chem 2007;53:766-72.
  • 16. Levine GN, Bates ER, Blankenship JC, et al. ACCF; AHA; SCAI. 2011 ACCF/AHA/SCAI guideline for percutaneous coronary intervention: executive summary: a report of the American college of cardiology foundation/american heart association task force on practice guidelines and the society for cardiovascular angiography and interventions. Catheter Cardiovasc Interv 2012;79:453-95.
  • 17. McCullough PA. Contrast-induced acute kidney injury. J Am Coll Cardiol. 2008;51:1419-28.
  • 18. Tumlin J, Stacul F, Adam A, et al. CIN Consensus working panel. Pathophysiology of contrast-induced nephropathy. Am J Cardiol 2006;98:14-20.
  • 19. James MT, Ghali WA, Knudtson ML, et al. Alberta provincial project for outcome assessment in coronary heart disease (APPROACH) Investigators. Associations between acute kidney injury and cardiovascular and renal outcomes after coronary angiography. Circulation 2011;123:409-16.
  • 20. Abaci O, Harmankaya O, Kocas B, et al. Long-term follow-up of patients at high risk for nephropathy after contrast exposure. Angiology 2015;66:514-8.
  • 21. Brunetti ND, Correale M, De Gennaro L, et al. Blunted inflammatory response in STEMI patients timely reperfused. J Cardiovasc Med (Hagerstown). 2014;15:48-52.
  • 22. Ruparelia N, Digby JE, Jefferson A, et al. Myocardial infarction causes inflammation and leukocyte recruitment at remote sites in the myocardium and in the renal glomerulus. Inflamm Res 2013;62:515-25.
  • 23. Watanabe M, Saito Y, Aonuma K, et al. CINC-J study investigators. Prediction of contrast-induced nephropathy by the serum creatinine level on the day following cardiac catheterization. J Cardiol 2016;68:412-418.
  • 24. Slocum NK, Grossman PM, Moscucci M, et al. The changing definition of contrast-induced nephropathy and its clinical implications: insights from the Blue Cross Blue Shield of Michigan Cardiovascular Consortium (BMC2). Am Heart J 2012;163:829-34.
  • 25. Farhan S, Vogel B, Tentzeris I, et al. Contrast induced acute kidney injury in acute coronary syndrome patients: A single centre experience. Eur Heart J Acute Cardiovasc Care 2016;5:55-61.
Annals of Medical Research-Cover
  • Yayın Aralığı: 12
  • Yayıncı: İnönü Üniversitesi Tıp Fakültesi
Sayıdaki Diğer Makaleler

Are we on the side of over-diagnosis and treatment inBI-RADS 4A breast lesions?

Tevfik Avcı, Hale Turnaoğlu, Murathan Erkent, Pelin Börcek, Pelin Kaya, Emre Karakaya

Unexpected location of hydatid cyst

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CLP1 associated pontocerebellar hypoplasia

Gül Demet KAYA ÖZÇORA, Dilek AKTAŞ, Sefer KUMANDAŞ

Changes in maxillary perfusion following Le Fort Iosteotomy in orthognathic surgery

Bahadır SANCAR, Burak ÜNLÜTÜRK, Ferhat MUSULLUOĞLU

A case of cardiac cyst hydatid with multiple systeminvolvement

Sait Murat DOĞAN, Özlem ELKIRAN, Gökalp OKUT, Cemşit KARAKURT, Ahmet SIĞIRCI, Sevgi TAŞOLAR, Güleç Mert DOĞAN

Antimicrobial activity of sapindus mukorossi andsaponaria officinalis extracts on streptococcus mutansand enterococcus faecalis

Meltem Mert Eren, Hüseyin Servi, Günçe Ozan, Benin Dikmen, Cansu Vatansever, Hulki Caner Yeğin

The effect of physical activity performed in the firsttrimester on the development of preeclampsia andgestational diabetes

Mehmet Murat Isikalan, Ali Acar, Haççe Yeniçeri

Disseminated mycobacterium avium complex infectionmimicking lymphoproliferative disorder in a kidneytransplant recipient

Didem TURGUT, Binnaz Handan ÖZDEMİR, Cemile Cansu ALPEREN, Mehmet HABERAL

Letter to the editor: Undetectable pancreatic insulinomaduring surgery

Ali Emre ATICI

A retrospective evaluation of restorations performed at therestorative dental treatment clinic of the dentistry facultyin Adiyaman university

Ömer Çellik