Saf duyusal inme sendromu şeklinde ortaya çıkan talamik bölge serebrovasküler hastalığı olgu sunumu

Saf duyusal inme, derin ya da yüzeyel duyuların ayrı ayrı veya beraber tutulduğu, duysal veya irritatif bozukluklar ile kendini gösteren klinik sendromdur. Bu sendromda en sık talamusun ventro-posterior nükleusu etkilenir. Talamik etkilenmeleri olan hastalarda yukarı bakış felci, hemiparezi, hemihipoestezi, konfüzyon, uykuya meyil, duygusal labilite olabilmektedir. Talamus tüm duyu modalitelerinin (koku hariç) ortak kesişim noktası olduğundan tat, işitme, dokunma ve ağrı gibi duyusal modaliteler saf duyusal inmelerde etkilenebilmektedir. 56 yaşında erkek hasta sol kol, bacak, yüz ve gövde yarısında uyuşma yakınması ile başvurdu. Manyetik rezonans görüntülemede sağ talamusta iskemi ile uyumlu lezyon belirlendi. Sonrasında santral nörojenik ağrı sendromu gelişti. Nadir görülmesi, ek belirti olmadan hemihipoestezi ile başvurabilmesi ve hızla tanınıp tedavi başlanması gerektiğinden sunulmaya değerli görülmüştür.

Deep or superficial senses are affected separately or together in pure sensory stroke. Sensory or irritative disturbances are characterized in this clinical syndrome. The most common posterior nucleus of thalamus ventro-posterior is affected. Paresis, hemiparesis, hemihypoesthesia, confusion, lethargy, emotional lability may be. All the senses (except the smell) intersect in the thalamus. Sensory modalities such as taste, hearing, touch, and pain can be affected in sensory stroke. 56 years old male patient presented with complaints of numbness in the left arm, leg, face and trunk. Magnetic resonance imaging revealed a lesion compatible with ischemia in the right thalamus. Subsequently, central neurogenic pain syndrome developed. It is rarely seen. It can be applied with hemihyphoesthesia without additional symptom. It is very valuable to be present because it is important to be known quickly and to start treatment.

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1. Fisher CM. Pure sensory stroke involving face, arm, and leg. Neurology 1965;15:76-80.

2. Combarros O, Polo JM, Pascual J, Berciano J. Evidence of somatotopic organization of the sensory thalamus based on infarction in the nucleus ventralis posterior. Stroke 1991;22:1445-1447.

3. Alstadhaug KB, Prytz JF. Pure sensory syndromes and post-stroke pain secondary to bilateral thalamic lacunar infarcts: a case report. Journal of medical case reports 2012;6:359.

4. Arboix A, García-Plata C, García-Eroles L, Massons J, Comes E, Oliveres M, Targa C. Clinical study of 99 patients with pure sensory stroke. Journal of neurology 2005;252: 156-162.

5. Caplan LR, Pessin MS, Mohr JP. Vertebrobasilar Occlusive Disease. Barnett HJM, Mohr JP, Stein BM, Yatsu FM, ed. Stroke Pathophysiology, Diagnosis, and Management. London: Churchill Livingstone;1992:443-515.

6. Barth A, Bogousslavsky J, Caplan LR. Thalamic Infarcts and Hemorrhages. Stroke Syndromes. Cambridge University Press;1995:276-284.

7. Schmahmann JD. Vascular syndromes of the thalamus. Stroke 2003;34:2264-78.

8. Arboix A, Padilla I, Massons J, García-Eroles L, Comes E, Targa C. Clinical study of 222 patients with pure motor stroke. Journal of Neurology, Neurosurgery & Psychiatry 2002;71:239-42.

9. Kim JS. Pure sensory stroke: clinical-radiological correlates of 21 cases. Stroke 1992; 23:983-7.

10. Klit H, Finnerup NB, Jensen TS. Central post-stroke pain: clinical characteristics, pathophysiology, and management. Lancet Neurol 2009;8:857-68.

11. Paciaroni M, Bogousslavsky J. Pure sensory syndromes in thalamic stroke. Eur Neurol 1998;39:211-7.

12. Schonewille WJ, Tuhrim S, Singer MB, Atlas SW. Diffusion-weighted MRI in acute lacunar syndromes. Stroke 1999;30:2066-9.

13. Bennett MI, Attal N, Backonja MM, Baron R, Bouhassira D, Freynhagen R, Jensen TS. Using screening tools to identify neuropathic pain. Pain 2007;127:199-203.

14. Kumral E, Deveci EE, Colak AY, Çağında AD, Erdoğan C. Multiple variant type thalamic infarcts: pure and combined types. Acta Neurologica Scandinavica 2015;131:102-10.